Breaking Research Decodes the Mystery of “The Rubbery White Clots”
First time, comprehensively characterised the anomalous intravascular casts (AICs),
commonly reported by embalmers worldwide as strange, rubbery white clots.
Research, significantly funded by New Zealand Doctors Speaking Out with Science (NZDSOS),
provides definitive analysis that these structures are a previously unrecognised and abnormal form of intravascular clotting.
Since 2021, global reports,
from embalmers and some clinicians have described the retrieval of long, elastic, white fibrous structures from blood vessels,
distinct from ordinary post-mortem clots.
New three-part study,
using international labs on three continents,
describes their structure, elemental composition and protein makeup.
Concluding they represent a novel and persistent pathological entity
Key Findings of the Trilogy:
Paper 1: Morphology & Histology
https://www.preprints.org/manuscript/…
Established that AICs are not ordinary clots.
They are elastic,
lumen-conforming,
branched structures that form under active blood flow
(shown by partial “Lines of Zahn”),
yet are strikingly devoid of intact red blood cells and platelets.
Their rubber-like consistency and cohesive strength are incompatible with known pre- and post-mortem changes.
Lines of Zahn
characteristic of thrombus formed at the site of rapid arterial blood flow,
with laminations produced by successive deposition of platelets and fibrin (pale layers),
alternating with red blood cells (dark layers).
Paper 2: Elemental Analysis
https://www.preprints.org/manuscript/…
Revealed the clots have a bizarre chemical fingerprint.
They are depleted in sulphur (a key marker of protein) and enriched in phosphorus,
a composition impossible for a normal, protein-dominant fibrin clot.
This indicates a hybrid organic-inorganic matrix, not a simple blood clot.
Paper 3: Proteomic Analysis
https://www.preprints.org/manuscript/…
Solved the protein puzzle.
While the clots do contain fibrinogen,
the building block of normal clots,
the fibrin chains are in a very abnormal ratio (~1:7:3 for α:β:γ chains vs. the normal 1:1:1).
Critically, they are almost completely lacking in plasminogen (the enzyme required to break down clots),
explaining their stubborn persistence.
The protein profile also shows signs of inflammatory and immune system involvement as well as red cell destruction.
Senior Researcher Dr Bruce Rapley
“This is not just a big blood clot. This is a fundamentally different architecture.
The profound deficiency in plasminogen is like building a structure impervious to future demolition – it’s designed to persist.
The elemental data confirms it’s not just protein; it’s a hybrid material our bodies are forced to make but not equipped to clear.”
This holds a significant health implication. The researchers note that the formation of such persistent, obstructing material in blood vessels, particularly if in the microvasculature, will lead to chronic oxygen lack, organ damage, pain, exhaustion, and cascades of inflammatory pathology.
The study concludes that AICs anomalous intravascular casts, “provide a mechanistically coherent explanation for persistent vascular obstruction,
impaired tissue perfusion,
inflammation, and a broad spectrum of acute and chronic organ dysfunction.”
A Call for Urgent Investigation:
The paper highlights the covid injections as a crucial research direction:
“If spike protein were demonstrated to provoke anomalous intravascular casts,
this would raise serious implications not only for covid pathophysiology but also for genetic platforms that induce sustained host manufacture of spike protein,
making it imperative that this potential association be rigorously investigated.”
Dr Shelton
“This analysis puts substance to the observations our organisation has been highlighting for 4 years now,”
“These are not ‘normal’ clots.
This work adds to the scientific basis for the persistent symptoms and deaths since the rollouts,
and strengthens our many calls to halt the covid injections pending further investigation.
We thank supporters for enabling this work and urge the global medical community to take these findings seriously.
Already these results are enabling rapid strides in showing how these harmful structures were predictable from first principles.”
The scientific papers are available on the preprint server and at www.nzdsos.com for review.
Youtube Transcript
0:01
You are most welcome to this video. Now,
0:03
it looks like we’re getting somewhere
0:04
with the science of the new rubbery
0:07
white blood clots that first reared
0:10
their ugly heads in 20 21 as far as we
0:15
know. We couldn’t find any records of
0:17
them before that. Perhaps a few in 2020,
0:19
but basically the pandemic of these
0:22
things started in 2021.
0:25
Now, I’m not going to be showing
0:26
pictures on this particular video. We
0:28
have looked at pictures in the past.
0:30
What I’m going to do is I’m going to put
0:31
another video up shortly that shows
0:33
nothing but pictures, but I don’t want
0:35
to put pictures on this one just in case
0:37
that flags it up because I have had a
0:39
problem with that in the past. Now, this
0:42
information has come from reputable
0:44
sources here. This is the New Zealand
0:47
doctor’s site. This is their press
0:50
release. Breaking research decodes the
0:52
mystery of the rubbery white clot. Now
0:54
of course that’s quite a bold claim. So
0:57
we need the evidence and we do have the
0:59
evidence here. It’s in the form of three
1:01
papers. This is the first paper here. Uh
1:05
morphological and hytoological
1:07
characteristics of anomalous
1:09
intravascular casts. Um now there is a
1:12
bit of terminology here but I’m not
1:14
going to do it in this video. So
1:16
morphology of course you know that just
1:17
means the shape the hisystologology is
1:19
is the tissue type that it’s made up of
1:22
and it’s characterization of anomalous
1:24
intravascular casts and we know this is
1:27
the case because these casts the these
1:30
white rubbery clots follow the shape of
1:32
the internal blood vessels. They’re
1:34
deposited inside the blood vessels and
1:36
and show the shape of these blood
1:38
vessels. And we’ve actually seen
1:40
pictures of they’ve been taken out of of
1:42
these clots being taken out of blood
1:45
vessels. So that’s the first paper
1:47
there. Obviously we can’t do them all in
1:49
detail today. Uh the second paper here
1:53
uh elemental characterization says this
1:55
is about the chemistry. The the the
1:58
makeup of them of anomalous
2:01
intravascular cast reveals an abnormal
2:04
biochemical matrix. And as we’ll see,
2:07
there are some rather strange features
2:09
of the chemistry of it that make it
2:11
difficult to explain. Although they do
2:13
put forward some good hypotheses on
2:15
that.
2:17
And the third paper is here. Proteinomic
2:20
characterization. Of course,
2:22
proteinomics is is the protein that is
2:24
made up of protein characterization
2:27
of anomalous intravascular cast reveals
2:30
non-canetical fibbrin architecture and
2:33
impaired fibbrinolyis. Uh what what this
2:37
means is non-initical meat basically
2:39
means it’s not in the cannon of
2:40
knowledge. This is this is new. This is
2:43
not in the recognized pathology
2:44
textbooks and and the we’ll look at the
2:47
fibbrin and the fibbrronogen as as we go
2:50
through.
2:52
So here’s the paper that we’ve seen from
2:54
the New Zealand doctors and that’s the
2:56
direct reference for that. It’s
2:57
available in the public domain.
3:01
So they’re saying first time that
3:03
they’ve comprehensively characterized
3:05
the anomalous intravascular casts and uh
3:08
I have been following this Roy for some
3:11
time now and I agree this is the first
3:13
full characterization that I’ve seen. In
3:16
fact the
3:18
silence from the pathological and
3:20
medical journals around the world on
3:22
this has been quite deafening.
3:25
Why aren’t they talking about this
3:26
strange new phenomena and racing to
3:29
characterize it? It really is quite
3:30
bizarre. It’s almost as if it’s been
3:33
edited out of the mainstream medical
3:35
journals.
3:38
Anyway, let’s carry on with what they’ve
3:39
got here. Commonly reported by inbalamas
3:42
worldwide as strange at rubbery white
3:44
clots and of course we have talked to
3:46
inbalamas worldwide, Richard Hirschman
3:48
in the states, John Oloney in the United
3:51
Kingdom and of course um m Major Tom
3:54
Havland has done a lot of research on
3:56
this as well.
3:58
Um, research significantly funded by New
4:01
Zealand doctors speaking out with
4:02
science. Great title.
4:05
Good for them. Uh, provides definitive
4:08
analysis that these structures are a
4:10
previously unrecognized and abnormal
4:12
form of intravascular clotting. Now,
4:17
this is consistent with all the imbalas
4:19
I’ve talked to and the pathologists.
4:21
I’ve talked to doctors and pathologists
4:23
about this as well. Some professors of
4:24
medicine about this and they haven’t
4:26
seen these before. These are not normal
4:29
uh permortem plots. The the these are
4:32
these are something new. Uh basically
4:35
2021 they seem to pop up that they seem
4:38
to be a few in 2020 but basically they
4:40
popped up in mostly in numbers any at
4:42
least in 2021.
4:46
um global reports from imbalmeras and
4:48
some clinicians have described the
4:50
retrieval of long elastic white fibrous
4:52
structures from blood vessels distinct
4:55
from ordinary postmortem clots. The
4:58
embombers who’ve been doing this for
4:59
decades tell us this is something new.
5:02
They’re used to what they call I think
5:04
it’s chicken clots they call them like
5:06
blood clots. They’re not like that.
5:08
These are white and they’re rubbery and
5:09
they have tensile strength.
5:12
This is something new now. Now, it’s a
5:15
new three-part study using international
5:16
labs on three continents. Sounds good.
5:19
Sounds good. Describes the structure,
5:21
elemental composition, and protein
5:22
makeup, concluding they represent a
5:24
novel and persistent pathological
5:28
entity. Now, um, Major Tom Havlin did
5:31
give us some data on the prevalence of
5:34
these clots from the limited amount of
5:37
survey data we could get, but he did
5:39
have hundreds of data points. And it
5:41
looks like these clots are getting
5:42
slightly less common, but they are still
5:45
there. And there’s something that’s
5:47
really frightening I’m going to tell you
5:48
about in a minute as well that they’re
5:51
still presenting as as a ongoing
5:54
phenomena albeit with a lower prevalence
5:58
or incident than they had.
6:01
Now key findings of the trilogy. This is
6:04
just the outline we’re going to give
6:05
here. Morphology and hisystologology. So
6:08
what’s the shape? What’s the tissue?
6:10
That’s the link there for the paper.
6:12
established that the uh anomalous
6:15
intravascular clots are not ordinary
6:17
clots. They are elastic lumen
6:19
conforming. In other words, they’re
6:21
casts. They form inside the vessel and
6:25
demonstrate the shape of the blood
6:28
vessels, branch structures that form
6:30
under active blood flow. Now, this is
6:34
shown partly by lines of XAN. Now, I’m
6:37
I’m going to tell you about those in a
6:38
minute. Um but basically this means that
6:41
these form during life
6:44
and if they form during life that means
6:47
they could be the cause of death which
6:49
is is quite a frightening idea really.
6:52
Up until I looked at these papers I knew
6:54
that these were some sort of permortm
6:56
phenomena. I assume they were caused as
6:58
part of the dying process but it’s
6:59
looking like not. And let me tell you
7:02
why I think that. Um you know just
7:06
before we do that that that they are
7:08
strikingly devoid of intact red blood
7:10
cells and platelets. Of course of course
7:13
ordinary blood clots are very rich in
7:15
red cells and platelets. Their rubbery
7:19
like consistency
7:21
and uh cohesive strength in other words
7:24
the hard to pull apart are incompatible
7:26
with known pre and postmortem changes.
7:30
Now these lines of Xan that they show
7:33
are characteristic of thrombus formed at
7:35
the site of rapid arterial blood flow.
7:38
So they’re formed when the blood is
7:40
still flowing with laminations produced
7:43
by successive deposits deposition of
7:46
platelets and fibbrin. So the fibbrin
7:48
layers are pale with uh darker layers
7:52
that are uh from the blood that are
7:54
darker layers. Now
7:56
this means that these formed when the
7:59
blood was still flowing. Therefore by
8:01
definition the person was still alive.
8:04
Therefore these clots may have been the
8:05
cause of death in many of these
8:09
individuals or at least some of these
8:10
individuals in which these clots are
8:12
found in the post-mortem and imbalming
8:17
situation. Um that was new to me and I
8:20
find that a disturbing finding.
8:24
Anyway, the second paper elemental
8:26
analysis
8:27
reveals that the clots have a bizarre
8:29
chemical fingerprint. They are depleted
8:31
in s uh they are uh depleted in sulfur,
8:36
a key marker of protein and enriched in
8:39
phosphorus. So what this means is um pro
8:44
proteins commonly contain sulfur of
8:45
course but not so much phosphorus. So,
8:48
they don’t have a a huge amount of
8:50
protein, but they do have a lot of
8:51
phosphorus, which is
8:55
rather rather strange. Again, um rather
8:59
difficult to explain. So, they’re
9:01
depleted in sulfur,
9:04
a key marker protein, and enriched in
9:06
phosphorus, a composition impossible for
9:09
a normal protein dominant fibbrin clot
9:12
because the fibbrin the the proteins
9:14
there would contain sulfur.
9:19
This indicates a hybrid organic
9:21
inorganic matrix. So, so in other words,
9:23
there’s some protein there that’s
9:25
organic. Organic simply means it
9:27
contains carbon, but some of it’s in
9:29
inorganic because the phosphorus is in
9:31
itself inorganic unless it’s combined
9:34
with carbon. So, this is not a simple
9:36
blood clot.
9:39
So, um in a sense here here really the
9:42
mystery deepens.
9:44
proteinomic analysis. Again, there’s the
9:47
link. They say they’ve solved the
9:49
protein puzzle. I’m not quite sure about
9:50
that. But, uh, while the clots do
9:53
contain fibbrinogen, the building blocks
9:55
of a normal clot, the fibbrin chains are
9:58
in very abnormal ratios 1:7 to three for
10:01
the alpha, beta, and gamma chains, not
10:02
the normal 1:1. So, the normal fibbrin
10:06
would have 1:1 alpha, beta, gamma
10:08
chains. But basically what’s happening
10:10
here is fibbrinogen produced in the
10:13
liver. It’s in solution in the blood.
10:15
When it precipitates out, the
10:16
fibbrronogen becomes these strands, this
10:18
sticky uh material. In in teaching
10:22
students, I said it was like
10:23
willow. I don’t know if you have in the
10:25
states, but it’s a it’s it’s a strand of
10:27
sticky stuff that we put on each other’s
10:29
clothes in summer. It kind of sticks.
10:32
And of course, the blood cells, the the
10:34
red blood cells stick to that forming
10:36
the clot.
10:37
So the fibbrinogen forms the fibbrin and
10:40
the fibbrin sticks the blood the blood
10:42
cells together to form the clot. But
10:45
it’s unusual fibbrin. It’s an abnormal
10:48
fibbrin
10:50
not what we would expect. Critically
10:53
they are almost completely lacking in
10:55
plasmminogen the enzyme required to
10:57
break down clots explaining their
10:59
stubborn persistence. So again uh just
11:02
the background here is that plasmminogen
11:05
is normally in solution in the plasma.
11:06
When there’s a blood clot that’s
11:08
converted to plasmine. The plasmine is
11:09
an enzyme which breaks down the blood
11:11
clot. But because these don’t have the
11:13
plasmminogen they don’t have the
11:15
plasmine. Therefore the clots are not
11:16
broken down.
11:18
So we’re getting these clots forming but
11:21
the normal mechanisms in the body that
11:23
break these clots down are absent.
11:26
Therefore, of course, the clots aren’t
11:28
broken down, explaining their stubborn
11:30
persistence.
11:32
The protein profile also so shows signs
11:35
of inflammatory and immune system
11:36
involvement. So, there’s inflammation
11:38
going on here. And it looks like it’s
11:42
got something to do with immunity.
11:45
There’s an abnormality of immunity going
11:48
on here as well. At least that’s what
11:52
these analysis would indicate.
11:56
um as well as red cell destruction. So,
11:59
senior research Bruce Rapley, one of the
12:01
doctors, this is not just a big blood
12:04
clot. This is a fundamentally different
12:06
architecture. We’re dealing with
12:07
something new here.
12:10
The profound efficiency in plasmminogen
12:12
is like a building building a structure
12:14
impervious to its future demolition. So,
12:16
there’s no plasmminogen to be converted
12:18
to to plasine to break the clot down.
12:20
So, it’s not broken down. It persists.
12:22
And we’ve seen great big long versions
12:24
of this that uh have been taken out of
12:27
blood vessels in the imbalming
12:29
situation.
12:31
It’s designed to persist or it persists.
12:37
The elemental data confirms it’s not
12:38
just protein. It’s a hybrid material our
12:40
bodies are forced to make but not
12:42
equipped to clear.
12:45
This holds a significant health
12:46
implication. The researchers note that
12:49
the formation of such persistent
12:51
obstructing material in blood vessels
12:54
particularly if the m in the
12:55
microvasculature will lead to chronic
12:57
oxygen lack or organ damage, pain,
12:59
exhaustion and cascades of inflammatory
13:04
pathology. And of course is the if
13:06
they’re big, it’s inconsistent with life
13:09
because it’s going to block off the
13:10
circulatory system.
13:13
again just making it so much more
13:15
surprising that this is not talked about
13:16
in the mainstream journals.
13:19
Um this can’t be hidden forever. It will
13:22
come out sooner or later. This is such
13:24
an obvious new pathology. It will come
13:26
out but but uh but not yet only in these
13:31
um publications
13:34
that make it public but are not
13:36
mainstream.
13:37
The study concludes that the uh the
13:40
clots the anomalous intravascular casts
13:43
AIC’s
13:45
uh provide a uh provide a
13:47
mechanistically coherent explanation for
13:50
persistent vascular obstruction
13:53
impaired tissue profusion inflammation
13:54
and a broad spectrum acute and chronic
13:56
organ dysfunction. And of course, I
13:59
would add uh
14:01
um it might make it so that some people
14:04
are no longer with us anymore.
14:07
Anyway, just to conclude the author’s uh
14:09
call for urgent action in investigation,
14:11
the paper highlights the co inject
14:14
injections as a crucial research
14:17
direction. So, this is something that
14:19
needs looked at crucially. There’s a
14:23
temporal correlation between the roll
14:25
out of the co vaccines and the first
14:30
identifi identification of these clots
14:32
and we could apply Bradford Hill
14:34
criteria to this quite readily if the
14:36
will was there to do so. If spike
14:39
protein were demonstrated to provoke
14:41
anomalous intravascular casts, this
14:43
would raise serious implications not
14:46
only for COVID pathophysiology but also
14:48
for genetic platforms that include
14:52
a sustained host manufacturer of spike
14:54
protein.
14:56
And we know that when you give the mRNA
14:59
vaccines,
15:00
it’s not possible to predict in a
15:02
particular individual how that will
15:05
systemically circulate. But we do we do
15:07
know it systemically circulates all
15:09
around the body coming in into contact
15:11
with all of the vascule or all the
15:14
endothelium lining the vascule of the
15:16
body. And of course we’re completely
15:18
unable to predict how long in any
15:21
individual how long spike protein will
15:23
be produced and how much spike protein
15:25
will be produced. The amount of spike
15:27
protein produced could vary by a factor
15:29
of a thousand
15:32
and we don’t know how long whether it’s
15:35
going to be days, weeks, months or uh
15:38
some studies are showing even longer
15:40
periods of time.
15:43
Uh making it uh imperative that this
15:47
potential association be rigorously
15:49
investigated. Dr. Sheldon, who we’ve had
15:51
on the channel before. Uh, this analysis
15:54
puts substance to the OB to to the
15:57
observations our organization has been
15:59
highlighting for four years now. Well
16:01
done to the New Zealand doctors. These
16:03
are not normal clots. This work adds to
16:06
the scientific basis for the persistent
16:08
symptoms and deaths since the rollouts.
16:12
I guess we know what he means the
16:14
rollouts of and strengthens our many
16:18
calls to halt the co injections pending
16:20
further investigations.
16:23
We thank supporters for enabling this
16:26
work and urge the global medical
16:29
community to take these findings
16:31
seriously.
16:34
Okay. And all the links are
16:38
there. So,
16:42
we’ve got papers now. Um, there’s no
16:45
excuse for the scientific community not
16:46
to act on this. And if mainstream
16:49
journals continue
16:52
to
16:54
um limit
16:57
their willingness to publish this sort
17:00
of material, uh, I think that their
17:04
ongoing validity is open to question.
17:08
We’ll leave that there. Have a look at
17:10
the papers yourself.
17:12
We are getting somewhere with this. Um,
17:15
this can’t be hidden for too much
17:16
longer. The science is accumulating. The
17:18
science is there now.
17:21
And, uh, thank you for watching. Just a
17:24
brief note. Um, if you see me, there’s
17:26
loads of channels have sprung up that
17:28
are cloning me now, these AI things. Um,
17:31
if you see me on any other YouTube
17:33
channel than this one, it’s not me. It’s
17:36
a scam. get don’t watch it. And you can
17:38
tell it’s my channel because it’s got
17:40
3.3 million subscribers. If it’s got 200
17:43
subscribers, it’s not me. And if you see
17:45
me on Facebook, that’s not me either.
17:49
Sadly, we’re moving into a world of deep
17:52
fakes.
17:53
But uh and you’ll always see me, not
17:56
just a picture of me. I’ll always be
17:57
talking on my videos. Um, so be aware of
18:02
scams and let’s hope
18:04
mainstream medical pathological academia
18:07
starts taking these rubbery clots
18:09
seriously. Another video shortly on the
18:12
pictures uh that I’m not putting on this
18:14
one just in case. But for now, thank you
18:16
for watching.